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ACAN

GeneName

ACAN

Summary

ACAN, also known as aggrecan, AGC1, or aggrecan core protein, is a large aggregating proteoglycan predominantly found in the extracellular matrix of cartilage and the central nervous system. With a mass of 261 kDa, it plays a vital role in providing structural support and resilience to tissues through its ability to bind water and form aggregates with hyaluronic acid. ACAN is involved in various biological processes, including cell adhesion, skeletal system development, and central nervous system development. It is also associated with the perineuronal net and synaptic structures, highlighting its importance in neural architecture and function.

Importance

ACAN is relevant to: - Cartilage health and integrity, influencing conditions such as osteoarthritis due to its role in maintaining the extracellular matrix. - Central nervous system function and development, which may have implications in neurodegenerative diseases. - Cell adhesion mechanisms, impacting tissue organisation and repair processes. - The regulation of proteolytic processes, which can affect various physiological and pathological states.

Top Products

For researchers investigating ACAN, we highly recommend the top-selling recombinant antibody, Anti-Aggrecan antibody [6-B-4] (ab3778). This well-cited product has garnered 261 citations, reflecting its strong reputation in the field. It has been validated for use in Western blotting (WB) and immunohistochemistry (IHC), making it a versatile choice for various applications. The recombinant nature of this antibody ensures batch-to-batch consistency, providing reliable results for your research on ACAN. The Anti-Aggrecan antibody - C-terminal ELISA Kit (ab140691) is an excellent option for researchers looking to measure ACAN levels in their samples.

Abcam Product Citation Summary

The data indicates that ACAN is primarily studied in the context of cartilage and chondrogenesis, particularly in human chondrocytes and articular cartilage. The use of various applications such as western blotting and immunohistochemistry highlights the importance of ACAN in understanding osteoarthritis progression and chondrogenic marker expression.

Abcam Product Citation Table

Product Code
Species
Application
Study Context
PMID
ab186414
Human
WB
Osteoarthritis progression
32732957
ab3778
Human
ICC
Articular chondrocytes
21192818
ab3778
Human
IHC
Micromass cultures
31842986
ab3778
Rat
IHC
Femoral articular cartilage samples
32455683
ab3778
Human
WB, ICC
Chondrogenic marker expression
32398124
ab3778
Human
WB
Chondrogenic differentiation
33167997

Developmental stage

Expression was detected in chondrocytes throughout the developing skeleton.

Domain

Two globular domains, G1 and G2, comprise the N-terminus of the proteoglycan, while another globular region, G3, makes up the C-terminus. G1 contains Link domains and thus consists of three disulfide-bonded loop structures designated as the A, B, B' motifs. G2 is similar to G1. The keratan sulfate (KS) and the chondroitin sulfate (CS) attachment domains lie between G2 and G3.

Function

This proteoglycan is a major component of extracellular matrix of cartilagenous tissues. A major function of this protein is to resist compression in cartilage. It binds avidly to hyaluronic acid via an N-terminal globular region.

Involvement in disease

Spondyloepiphyseal dysplasia type Kimberley

SEDK

Spondyloepiphyseal dysplasias are a heterogeneous group of congenital chondrodysplasias that specifically affect epiphyses and vertebrae. The autosomal dominant SEDK is associated with premature degenerative arthropathy.

None

The disease is caused by variants affecting the gene represented in this entry.

Spondyloepimetaphyseal dysplasia, aggrecan type

SEMDAG

A bone disease characterized by severe short stature, macrocephaly, severe midface hypoplasia, short neck, barrel chest and brachydactyly. The radiological findings comprise long bones with generalized irregular epiphyses with widened metaphyses, especially at the knees, platyspondyly, and multiple cervical-vertebral clefts.

None

The disease is caused by variants affecting the gene represented in this entry.

Short stature and advanced bone age, with or without early-onset osteoarthritis and/or osteochondritis dissecans

SSOAOD

An autosomal dominant disease characterized by short stature, advanced bone maturation, early-onset osteoarthritis, and mild dysmorphic features consisting of midface hypoplasia, brachydactyly, broad great toes, and lumbar lordosis. Other features include intervertebral disk disease and osteochondritis dissecans. Osteochondritis dissecans is defined as a separation of cartilage and subchondral bone from the surrounding tissue.

None

The disease is caused by variants affecting the gene represented in this entry.

Post-translational modifications

Contains mostly chondroitin sulfate, but also keratan sulfate chains, N-linked and O-linked oligosaccharides. The release of aggrecan fragments from articular cartilage into the synovial fluid at all stages of human osteoarthritis is the result of cleavage by aggrecanase.

Sequence Similarities

Belongs to the aggrecan/versican proteoglycan family.

Tissue Specificity

Detected in fibroblasts (at protein level) (PubMed:36213313). Restricted to cartilage (PubMed:7524681).

Cellular localization

Alternative names

AGC1, CSPG1, MSK16, ACAN, Aggrecan core protein, Cartilage-specific proteoglycan core protein, Chondroitin sulfate proteoglycan core protein 1, CSPCP, Chondroitin sulfate proteoglycan 1

swissprot:P16112 entrezGene:176 omim:155760