ab4808 を使用した論文を発表された方は、こちらまでお知らせください。データシートに掲載させていただきます。

ab4808 は 11 報の論文で使用されています。

  • Lemonidis K  et al. Identification of a Novel Sequence Motif Recognized by the Ankyrin Repeat Domain of zDHHC17/13 S-Acyltransferases. J Biol Chem 290:21939-50 (2015). PubMed: 26198635
  • Grefen C The split-ubiquitin system for the analysis of three-component interactions. Methods Mol Biol 1062:659-78 (2014). WB . PubMed: 24057391
  • Dittmer S  et al. TOX3 is a neuronal survival factor that induces transcription depending on the presence of CITED1 or phosphorylated CREB in the transcriptionally active complex. J Cell Sci 124:252-60 (2011). PubMed: 21172805
  • Bot N  et al. Processing of the Synaptic Cell Adhesion Molecule Neurexin-3{beta} by Alzheimer Disease {alpha}- and {gamma}-Secretases. J Biol Chem 286:2762-73 (2011). WB . PubMed: 21084300
  • Pan MH  et al. Monitoring molecular-specific pharmacodynamics of rapamycin in vivo with inducible Gal4->Fluc transgenic reporter mice. Mol Cancer Ther 9:2752-60 (2010). WB . PubMed: 20858726
  • Grefen C  et al. A novel motif essential for SNARE interaction with the K(+) channel KC1 and channel gating in Arabidopsis. Plant Cell 22:3076-92 (2010). WB . PubMed: 20884800
  • Grefen C  et al. The determination of protein-protein interactions by the mating-based split-ubiquitin system (mbSUS). Methods Mol Biol 479:217-33 (2009). WB . PubMed: 19083185
  • Arendt ML  et al. A novel two-step transcriptional activation system for gene therapy directed toward epithelial cells. Mol Cancer Ther 8:3244-54 (2009). WB . PubMed: 19952120
  • Holmberg J  et al. SoxB1 transcription factors and Notch signaling use distinct mechanisms to regulate proneural gene function and neural progenitor differentiation. Development 135:1843-51 (2008). IHC-P . PubMed: 18417619
  • Grefen C  et al. Split-ubiquitin system for identifying protein-protein interactions in membrane and full-length proteins. Curr Protoc Neurosci Chapter 5:Unit 5.27 (2007). WB . PubMed: 18428659
  • Yang Q  et al. Peroxisome proliferator-activated receptor alpha activation during pregnancy severely impairs mammary lobuloalveolar development in mice. Endocrinology 147:4772-80 (2006). IHC-P . PubMed: 16857745

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