Weyemi U et al.Histone H2AX deficiency causes neurobehavioral deficits and impaired redox homeostasis.Nat Commun 9:1526 (2018).
Zhang H et al.Combination of betulinic acid and chidamide inhibits acute myeloid leukemia by suppression of the HIF1a pathway and generation of reactive oxygen species.Oncotarget 8:94743-94758 (2017).
Liu R et al.ASCIZ/ATMIN is dispensable for ATM signaling in response to replication stress.DNA Repair (Amst) 57:29-34 (2017).
Xie W et al.Perinatal testosterone exposure potentiates vascular dysfunction by ERß suppression in endothelial progenitor cells.PLoS One 12:e0182945 (2017).
Zou Y et al.Prenatal levonorgestrel exposure induces autism-like behavior in offspring through ERß suppression in the amygdala.Mol Autism 8:46 (2017).
Yu H et al.Combination of betulinic acid and chidamide synergistically inhibits Epstein-Barr virus replication through over-generation of reactive oxygen species.Oncotarget 8:61646-61661 (2017).
Weyemi U et al.The histone variant H2A.X is a regulator of the epithelial-mesenchymal transition.Nat Commun 7:10711 (2016).
WB, ChIP, ICC/IF
Weyemi U et al.Twist1 and Slug mediate H2AX-regulated epithelial-mesenchymal transition in breast cells.Cell Cycle 15:2398-404 (2016).
Neumayer G & Nguyen MD TPX2 impacts acetylation of histone H4 at lysine 16: implications for DNA damage response.PLoS One 9:e110994 (2014).
Zalmas LP et al.E2F-7 couples DNA damage-dependent transcription with the DNA repair process.Cell Cycle 12:3037-51 (2013).
Lee JH et al.Histone deacetylase inhibitor induces DNA damage, which normal but not transformed cells can repair.Proc Natl Acad Sci U S A 107:14639-44 (2010).